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Associations between maternal genotypes and metabolites implicated in congenital heart defects

CITE Title : Associations between maternal genotypes and metabolites implicated in congenital heart defects Personal Author(s) : Chowdhury, Shimul;Hobbs, Charlotte A.;MacLeod, Stewart L.;Cleves, Mario A.;Melnyk, Stepan;James, S. Jill;Hu, Ping;Erickson, Stephen W.; Published Date : September 27 2012;11-2012; Source : Mol Genet Metab. 2012; 107(3):596-604 URL : https://stacks.cdc.gov/view/cdc/33495 Chowdhury, Shimul et al. (2012). Associations between maternal genotypes and metabolites implicated in congenital heart defects. 107(3). Chowdhury, Shimul et al. "Associations between maternal genotypes and metabolites implicated in congenital heart defects" 107, no. 3 (2012) Chowdhury, Shimul et al. "Associations between maternal genotypes and metabolites implicated in congenital heart defects" vol. 107, no. 3, 2012 Export RIS Citation Information.

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Chowdhury, Shimul ;

Hobbs, Charlotte A.

September 27 2012 | Mol Genet Metab. 2012; 107(3):596-604

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BackgroundThe development of non-syndromic congenital heart defects (CHDs) involves a complex interplay of genetics, metabolism, and lifestyle. Previo...
Completeness of required site-specific factors for brain and CNS tumors in the Surveillance, Epidemiology and End Results (SEER) 18 database (2004-2012, varying)

CITE Title : Completeness of required site-specific factors for brain and CNS tumors in the Surveillance, Epidemiology and End Results (SEER) 18 database (2004-2012, varying) Personal Author(s) : Ostrom, Quinn T.;Gittleman, Haley;Kruchko, Carol;Louis, David N.;Brat, Daniel J.;Gilbert, Mark R.;Petkov, Valentina I.;Barnholtz-Sloan, Jill S.; Published Date : July 14 2016 Source : J Neurooncol. 130(1):31-42 URL : https://stacks.cdc.gov/view/cdc/51019 Ostrom, Quinn T. et al. (2016). Completeness of required site-specific factors for brain and CNS tumors in the Surveillance, Epidemiology and End Results (SEER) 18 database (2004-2012, varying). 130(1). Ostrom, Quinn T. et al. "Completeness of required site-specific factors for brain and CNS tumors in the Surveillance, Epidemiology and End Results (SEER) 18 database (2004-2012, varying)" 130, no. 1 (2016) Ostrom, Quinn T. et al. "Completeness of required site-specific factors for brain and CNS tumors in the Surveillance, Epidemiology and End Results (SEER) 18 database (2004-2012, varying)" vol. 130, no. 1, 2016 Export RIS Citation Information.

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Ostrom, Quinn T. ;

Gittleman, Haley

July 14 2016 | J Neurooncol. 130(1):31-42

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Cancer registries are an important source of population-level information on brain tumor incidence and survival. Surveillance, Epidemiology, and End R...
Myxoid glioneuronal tumor of the septum pellucidum and lateral ventricle is defined by a recurrent PDGFRA p.K385 mutation and DNT-like methylation profile

CITE Title : Myxoid glioneuronal tumor of the septum pellucidum and lateral ventricle is defined by a recurrent PDGFRA p.K385 mutation and DNT-like methylation profile Personal Author(s) : Solomon, David A.;Korshunov, Andrey;Sill, Martin;Jones, David T.W.;Kool, Marcel;Pfister, Stefan M;Fan, Xuemo;Bannykh, Serguei;Hu, Jethro;Danielpour, Moise;Li, Rong;Johnston, James;Cham, Elaine;Cooney, Tabitha;Sun, Peter;Bush, Nancy Ann Oberheim;McDermott, Michael;Van Ziffle, Jessica;Onodera, Courtney;Grenert, James P.;Bastian, Boris C.;Villanueva-Meyer, Javier E.;Pekmezci, Melike;Bollen, Andrew W.;Perry, Arie; Published Date : July 13 2018 Source : Acta Neuropathol. 136(2):339-343 URL : https://stacks.cdc.gov/view/cdc/100398 Solomon, David A. et al. (2018). Myxoid glioneuronal tumor of the septum pellucidum and lateral ventricle is defined by a recurrent PDGFRA p.K385 mutation and DNT-like methylation profile. 136(2). Solomon, David A. et al. "Myxoid glioneuronal tumor of the septum pellucidum and lateral ventricle is defined by a recurrent PDGFRA p.K385 mutation and DNT-like methylation profile" 136, no. 2 (2018) Solomon, David A. et al. "Myxoid glioneuronal tumor of the septum pellucidum and lateral ventricle is defined by a recurrent PDGFRA p.K385 mutation and DNT-like methylation profile" vol. 136, no. 2, 2018 Export RIS Citation Information.

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Solomon, David A. ;

Korshunov, Andrey

July 13 2018 | Acta Neuropathol. 136(2):339-343

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Minor changes in expression of the mismatch repair protein MSH2 exert a major impact on glioblastoma response to temozolomide

CITE Title : Minor changes in expression of the mismatch repair protein MSH2 exert a major impact on glioblastoma response to temozolomide Personal Author(s) : McFaline-Figueroa, José L.;Braun, Christian J.;Stanciu, Monica;Nagel, Zachary D.;Mazzucato, Patrizia;Sangaraju, Dewakar;Cerniauskas, Edvinas;Barford, Kelly;Vargas, Amanda;Chen, Yimin;Tretyakova, Natalia;Lees, Jacqueline A.;Hemann, Michael T.;White, Forest M.;Samson, Leona D.; Published Date : May 29 2015 Source : Cancer Res. 75(15):3127-3138. URL : https://stacks.cdc.gov/view/cdc/34825 McFaline-Figueroa, José L. et al. (2015). Minor changes in expression of the mismatch repair protein MSH2 exert a major impact on glioblastoma response to temozolomide. 75(15). McFaline-Figueroa, José L. et al. "Minor changes in expression of the mismatch repair protein MSH2 exert a major impact on glioblastoma response to temozolomide" 75, no. 15 (2015) McFaline-Figueroa, José L. et al. "Minor changes in expression of the mismatch repair protein MSH2 exert a major impact on glioblastoma response to temozolomide" vol. 75, no. 15, 2015 Export RIS Citation Information.

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McFaline-Figueroa, José L. ;

Braun, Christian J.

May 29 2015 | Cancer Res. 75(15):3127-3138.

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Glioblastoma (GBM) is often treated with the cytotoxic drug temozolomide, but the disease inevitably recurs in a drug-resistant form after initial tre...