Details:

Alternative Title:
Prev Chronic Dis
Personal Author:
Haemer, Matthew A. ; Huang, Terry T. ; Daniels, Stephen R.
Haemer, Matthew A. ; Huang, Terry T. ; Daniels, Stephen R. Less -
Description:
Molecular, cellular, and epidemiologic findings suggest that neurohormonal, epigenetic, and microbiologic mechanisms may influence risk for obesity by interacting with socioenvironmental factors. Homeostatic and nonhomeostatic neural controls of energy predispose people to obesity, and this predisposition may be exaggerated by the influence of media, marketing, and sleep patterns. Epigenetic gene regulation may account for the influence of modifiable early life or maternal exposures on obesity risk. Alterations in gut flora caused by infant feeding practices or later diet may influence the absorption and storage of energy. Further exploration of how these molecular-cellular mechanisms might increase obesity risk in response to modifiable socioeconomic factors requires the partnership of laboratory and public health researchers.
Subjects:
[+]
Animals Epigenesis, Genetic Gastrointestinal Tract Homeostasis Humans Mice Models, Animal Models, Biological Neurotransmitter Agents Obesity Rats Risk Factors
Source:
Prev Chronic Dis. 6(3).
Document Type:
Journal Article
Genre:
Special Topic
Collection(s):
Preventing Chronic Disease
Main Document Checksum:
[+]
urn:sha256:a6995272a6073178aa2d0f3ceffc752f5e234212fd57c3c855c6aeb8fed3e506
Download URL:
https://stacks.cdc.gov/view/cdc/20386/cdc_20386_DS1.pdf
File Type:

Supporting Files

• 	license.txt	txt
  	PCD63A96.nxml	txt

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