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Siloxane nanoprobes for labeling and dual modality imaging of neural stem cells

CITE Title : Siloxane nanoprobes for labeling and dual modality imaging of neural stem cells Personal Author(s) : Addington, Caroline P.;Cusick, Alex;Shankar, Rohini Vidya;Agarwal, Shubhangi;Stabenfeldt, Sarah E.;Kodibagkar, Vikram D.; Published Date : Nov 23 2015 Source : Ann Biomed Eng. 44(3):816-827. URL : https://stacks.cdc.gov/view/cdc/40476 Addington, Caroline P. et al. (2015). Siloxane nanoprobes for labeling and dual modality imaging of neural stem cells. 44(3). Addington, Caroline P. et al. "Siloxane nanoprobes for labeling and dual modality imaging of neural stem cells" 44, no. 3 (2015) Addington, Caroline P. et al. "Siloxane nanoprobes for labeling and dual modality imaging of neural stem cells" vol. 44, no. 3, 2015 Export RIS Citation Information.

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Addington, Caroline P. ;

Cusick, Alex

Nov 23 2015 | Ann Biomed Eng. 44(3):816-827.

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Cell therapy represents a promising therapeutic for a myriad of medical conditions, including cancer, traumatic brain injury, and cardiovascular disea...
Enhancing neural stem cell response to SDF-1α gradients through hyaluronic acid-laminin hydrogels

CITE Title : Enhancing neural stem cell response to SDF-1α gradients through hyaluronic acid-laminin hydrogels Personal Author(s) : Addington, CP;Heffernan, JM;Millar-Haskell, CS;Tucker, EW;Sirianni, RW;Stabenfeldt, SE; Published Date : August 25 2015 Source : Biomaterials. 72:11-19 URL : https://stacks.cdc.gov/view/cdc/36971 Addington, CP et al. (2015). Enhancing neural stem cell response to SDF-1α gradients through hyaluronic acid-laminin hydrogels. 72 Addington, CP et al. "Enhancing neural stem cell response to SDF-1α gradients through hyaluronic acid-laminin hydrogels" 72 (2015) Addington, CP et al. "Enhancing neural stem cell response to SDF-1α gradients through hyaluronic acid-laminin hydrogels" vol. 72, 2015 Export RIS Citation Information.

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Addington, CP ;

Heffernan, JM

August 25 2015 | Biomaterials. 72:11-19

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Traumatic brain injury (TBI) initiates an expansive biochemical insult that is largely responsible for the long-term dysfunction associated with TBI; ...