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The Impact of Family and Peer Protective Factors on Girls’ Violence Perpetration and Victimization
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September 27 2012
Source: J Adolesc Health. 2012; 52(3):365-371
[PDF-372.15 KB]
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Alternative Title:J Adolesc Health
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Description:Purpose
This study investigates whether family and peer connections and prosocial norms buffer adolescent girls’ violence involvement, and whether a youth development intervention augments the power of these protective factors in reducing girls’ risk for violence.
Methods
Data were obtained from 253 13–17 year-olds enrolled in a randomized controlled trial of Prime Time, a youth development intervention offered through urban clinic settings to girls at high risk for pregnancy. Participants completed an A-CASI survey at baseline, 6, 12, and 18 months following enrollment. Protective factors included scales assessing family and peer connections and prosocial norms. Outcome variables were violence victimization and perpetration scales measured at 18 months.
Results
Family connections and prosocial norms independently protected girls against violence involvement. Peer prosocial norms also served as a protective buffer against violence perpetration and victimization; however, girls with strong peer connections had higher levels of violence perpetration. Participation in Prime Time augmented the protective effects of family and peer connections on girls’ violence victimization but not perpetration. Prime Time participants who had high levels of family connections reported the lowest levels of violence victimization at 18 months. Prime Time participants with strong peer connections trended toward lower levels of violence victimization than other girls.
Conclusions
Results suggest that effects of the Prime Time intervention on violence victimization were optimized among high-risk adolescent girls with strong connections to family and peers. The intervention was most potent in preventing violence victimization among girls with strong prosocial connections to family and peers.
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Subject:
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Pubmed ID:23299002
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Pubmed Central ID:PMC3579999
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