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Farnesyltransferase Inhibition Exacerbates Eosinophilic Inflammation and Airway Hyperreactivity in Mice with Experimental Asthma: The Complex Roles of Ras GTPase and Farnesylpyrophosphate in Type-2 Allergic Inflammation

CITE Title : Farnesyltransferase Inhibition Exacerbates Eosinophilic Inflammation and Airway Hyperreactivity in Mice with Experimental Asthma: The Complex Roles of Ras GTPase and Farnesylpyrophosphate in Type-2 Allergic Inflammation Personal Author(s) : Bratt, Jennifer M.;Chang, Kevin Y.;Rabowsky, Michelle;Franzi, Lisa M.;Ott, Sean P.;Filosto, Simone;Goldkorn, Tzipora;Arif, Muhammad;Last, Jerold A.;Kenyon, Nicholas J.;Zeki, Amir A.; Published Date : 6 01 2018;6-01-; Source : J Immunol. 200(11):3840-3856 URL : https://stacks.cdc.gov/view/cdc/55074 Bratt, Jennifer M. et al. (2018). Farnesyltransferase Inhibition Exacerbates Eosinophilic Inflammation and Airway Hyperreactivity in Mice with Experimental Asthma: The Complex Roles of Ras GTPase and Farnesylpyrophosphate in Type-2 Allergic Inflammation. 200(11). Bratt, Jennifer M. et al. "Farnesyltransferase Inhibition Exacerbates Eosinophilic Inflammation and Airway Hyperreactivity in Mice with Experimental Asthma: The Complex Roles of Ras GTPase and Farnesylpyrophosphate in Type-2 Allergic Inflammation" 200, no. 11 (2018) Bratt, Jennifer M. et al. "Farnesyltransferase Inhibition Exacerbates Eosinophilic Inflammation and Airway Hyperreactivity in Mice with Experimental Asthma: The Complex Roles of Ras GTPase and Farnesylpyrophosphate in Type-2 Allergic Inflammation" vol. 200, no. 11, 2018 Export RIS Citation Information.

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Bratt, Jennifer M. ;

Chang, Kevin Y.

6 01 2018 | J Immunol. 200(11):3840-3856

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Ras, a small GTPase protein, is thought to mediate Th2-dependent eosinophilic inflammation in asthma. Ras requires cell membrane association for its b...
Animal Models and Mechanisms of Tobacco Smoke-induced Chronic Obstructive Pulmonary Disease (COPD)

CITE Title : Animal Models and Mechanisms of Tobacco Smoke-induced Chronic Obstructive Pulmonary Disease (COPD) Personal Author(s) : Upadhyay, Priya;Wu, Ching-Wen;Pham, Alexa;Zeki, Amir A.;Royer, Christopher M.;Kodavanti, Urmila P.;Takeuchi, Minoru;Bayram, Hasan;Pinkerton, Kent E.; Published Date : 7 04 2023;7-04-; Source : J Toxicol Environ Health B Crit Rev. 26(5):275-305 URL : https://stacks.cdc.gov/view/cdc/137177 Upadhyay, Priya et al. (2023). Animal Models and Mechanisms of Tobacco Smoke-induced Chronic Obstructive Pulmonary Disease (COPD). 26(5). Upadhyay, Priya et al. "Animal Models and Mechanisms of Tobacco Smoke-induced Chronic Obstructive Pulmonary Disease (COPD)" 26, no. 5 (2023) Upadhyay, Priya et al. "Animal Models and Mechanisms of Tobacco Smoke-induced Chronic Obstructive Pulmonary Disease (COPD)" vol. 26, no. 5, 2023 Export RIS Citation Information.

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Upadhyay, Priya ;

Wu, Ching-Wen

7 04 2023 | J Toxicol Environ Health B Crit Rev. 26(5):275-305

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Chronic obstructive pulmonary disease (COPD) is the third leading cause of death worldwide, and its global health burden is increasing. COPD is charac...